Inflammatory mediators and acute infections.
In this paper some important mediation systems of inflammation are reviewed; special emphasis is laid on basic aspects of these systems, their interrelationships, and on clinically relevant effects with regard to the circulatory and pulmonary complications of sepsis. The references cited are selected on the basis of their review aspects.
D016718:Arachidonic Acid; D001095:Arachidonic Acids / Q000502:physiology; D003167:Complement Activation; D005174:Factor XII / Q000502:physiology; D006632:Histamine / Q000502:physiology; D006801:Humans; D007239:Infections / Q000503:physiopathology*; D007249:Inflammation / Q000503:physiopathology*; D009504:Neutrophils / Q000502:physiology; D010972:Platelet Activating Factor / Q000502:physiology; D011969:Receptors, Histamine H1 / Q000502:physiology; D011970:Receptors, Histamine H2 / Q000502:physiology; D012772:Shock, Septic / Q000503:physiopathology
D001095:Arachidonic Acids; D010972:Platelet Activating Factor; D011969:Receptors, Histamine H1; D011970:Receptors, Histamine H2; D016718:Arachidonic Acid; D006632:Histamine; D005174:Factor XII