A perspective on the treatment of aphakic cystoid macular edema.
Concepts related to the pathogenesis of aphakic cystoid macular edema (ACME) are presented with reference to possible management considerations. The role of certain aspects of cataract surgical technique, laser photocoagulation, vitreoretinal surgery and medical therapy utilizing corticosteroids and antiprostaglandins are discussed. The difficulties encountered in the evaluation of the efficacy of any treatment modality advocated for ACME are reviewed. Preliminary results of studies on the medical treatment of ACME currently under investigation by the author are also presented.
D000893:Anti-Inflammatory Agents / Q000627:therapeutic use*; D001095:Arachidonic Acids / Q000096:biosynthesis / Q000502:physiology; D002387:Cataract Extraction / Q000009:adverse effects; D002908:Chronic Disease; D005451:Fluorescein Angiography; D006801:Humans; D053685:Laser Therapy; D008269:Macular Edema / Q000175:diagnosis / Q000209:etiology / Q000378:metabolism / Q000628:therapy*; D011448:Prostaglandin Antagonists / Q000627:therapeutic use*; D011453:Prostaglandins / Q000096:biosynthesis* / Q000502:physiology; D014792:Visual Acuity; D014821:Vitrectomy
D000893:Anti-Inflammatory Agents; D001095:Arachidonic Acids; D011448:Prostaglandin Antagonists; D011453:Prostaglandins