[Pathology of labyrinthine hearing loss].
The most common causes auf sensorineural deafness lie in the cochlea. Other causes are less numerous, such that our presentation will be limited to cochleal disturbances. In a general introduction we shall attempt to analyse the structural reactions, metabolism and circulation of the cochlea and the behaviour of the inner ear lymph to a noxa. The monotony of the reactions will be described as well as the reversibility of some of the changes which are observed. All these reactions play a part in the normal functioning of the inner ear. The special part of the presentation will discuss the different forms of sensorineural deafness with which we are confronted daily, taking particular account of morphological and metabolic characteristics. We shall attempt to summarise wellknown material, and discuss newer information in greater detail. We shall pont out possible causes of sensorineural deafness which have seldomly been discussed.
D000328:Adult; D000368:Aged; D002675:Child, Preschool; D003051:Cochlea / Q000098:blood supply / Q000473:pathology; D003056:Cochlear Nerve / Q000473:pathology; D003638:Deafness / Q000473:pathology; D004734:Energy Metabolism; D006198:Hair Cells, Auditory / Q000473:pathology; D006317:Hearing Loss, Noise-Induced / Q000473:pathology; D006319:Hearing Loss, Sensorineural / Q000139:chemically induced / Q000235:genetics / Q000473:pathology*; D006801:Humans; D007223:Infant; D007231:Infant, Newborn; D007511:Ischemia / Q000473:pathology; D008854:Microscopy, Electron; D010040:Otosclerosis / Q000473:pathology; D011304:Presbycusis / Q000473:pathology
Pathologie der Innenohrschwerhörigkeiten.