Conversion from chronic to acute coronary artery disease: speculation regarding mechanisms.
D000328:Adult; D000368:Aged; D000787:Angina Pectoris / Q000209:etiology*; D000788:Angina Pectoris, Variant / Q000209:etiology; D000789:Angina, Unstable / Q000188:drug therapy / Q000209:etiology; D001011:Aorta / Q000378:metabolism; D001241:Aspirin / Q000627:therapeutic use; D001794:Blood Pressure; D003326:Coronary Circulation; D003327:Coronary Disease / Q000378:metabolism / Q000503:physiopathology*; D003331:Coronary Vessels / Q000378:metabolism; D011464:Epoprostenol / Q000378:metabolism; D005260:Female; D006439:Hemodynamics; D006801:Humans; D008297:Male; D008875:Middle Aged; D009203:Myocardial Infarction / Q000209:etiology* / Q000517:prevention & control; D010974:Platelet Aggregation; D011453:Prostaglandins / Q000378:metabolism; D013928:Thromboxane A2 / Q000378:metabolism; D013929:Thromboxane B2 / Q000378:metabolism
D011453:Prostaglandins; D013929:Thromboxane B2; D013928:Thromboxane A2; D011464:Epoprostenol; D001241:Aspirin