Renal glycogen content and hormonal control of enzymes involved in renal glycogen metabolism.
Fetal rat kidney showed glycogen deposition that reached a maximal value of 60 micrograms . mg prot-1 on day 18 and declined there-after. At birth glycogen concentration is reduced (20 micrograms . mg prot-1) but higher than adult one (cortex, 2.2 micrograms . mg prot-1 and medulla, 3.4 micrograms . mg prot-1). From day 17 to the birth, glycogen synthetase and phosphorylase activities increased slowly except for acid glucosidase activity which increased rapidly between day 18 to the birth (3-fold). Corticosteroid deprivation had no effect upon glycogen content but fetal decapitation on day 16 reduced glycogen content in kidney of 19-day-old fetuses.
D000305:Adrenal Cortex Hormones / Q000502:physiology; D000367:Age Factors; D000818:Animals; D000831:Animals, Newborn / Q000378:metabolism; D005333:Fetus / Q000378:metabolism; D005087:Glucan 1,4-alpha-Glucosidase / Q000378:metabolism; D006003:Glycogen / Q000378:metabolism*; D006006:Glycogen Synthase / Q000378:metabolism*; D006728:Hormones / Q000502:physiology*; D007028:Hypothalamic Hormones / Q000502:physiology; D007668:Kidney / Q000201:enzymology / Q000378:metabolism*; D006005:Phosphorylases / Q000378:metabolism*; D010907:Pituitary Hormones / Q000502:physiology; D051381:Rats
D000305:Adrenal Cortex Hormones; D006728:Hormones; D007028:Hypothalamic Hormones; D010907:Pituitary Hormones; D006003:Glycogen; D006005:Phosphorylases; D006006:Glycogen Synthase; D005087:Glucan 1,4-alpha-Glucosidase