The effects of group B streptococcus and other organisms on the pulmonary vasculature.
In this article, the authors review the effects of group B streptococcus, Pseudomonas aeruginosa, and Escherichia coli on the pulmonary vasculature and the possible mechanisms involved in their genesis.
D000380:Agranulocytosis / Q000209:etiology; D000403:Airway Resistance; D000818:Animals; D001427:Bacterial Toxins; D002199:Capillary Permeability; D004731:Endotoxins; D004926:Escherichia coli; D006976:Hypertension, Pulmonary / Q000209:etiology* / Q000378:metabolism / Q000503:physiopathology; D007213:Indomethacin / Q000494:pharmacology; D008168:Lung / Q000098:blood supply* / Q000378:metabolism / Q000473:pathology; D008196:Lymph / Q000502:physiology; D008775:Methylprednisolone / Q000494:pharmacology; D008833:Microcirculation; D011453:Prostaglandins / Q000096:biosynthesis; D011550:Pseudomonas aeruginosa; D011652:Pulmonary Circulation; D012756:Sheep; D013292:Streptococcus agalactiae; D014661:Vasoconstriction
D001427:Bacterial Toxins; D004731:Endotoxins; D011453:Prostaglandins; D008775:Methylprednisolone; D007213:Indomethacin