[Dependence of the cocarcinogenic action of the influenza virus on the nature of the influenza infection].
It was shown in experiments of CC57W mice that cocarcinogenic activity of influenza A/PR8/34 virus correlates with acute or chronic pattern of infection. Prolonged persistence of the virus resulted in significant stimulation of the lung tumor incidence in infected mice. The prevention with thymosin of chronic influenza infection development in CC57W mice lead to a decrease in the incidence of lung tumors to the control level.
D000818:Animals; D003043:Cocarcinogenesis; D009980:Influenza A virus / Q000472:pathogenicity*; D008175:Lung Neoplasms / Q000209:etiology* / Q000382:microbiology; D051379:Mice; D009976:Orthomyxoviridae Infections / Q000150:complications* / Q000188:drug therapy / Q000382:microbiology; D013947:Thymosin / Q000627:therapeutic use; D013997:Time Factors
D013947:Thymosin
Zavisimosti kokantserogennogo deĭstviia virusa grippa ot kharaktera grippoznoĭ infektsii.