[Functional state of mitochondria during early burn toxemia and exposure to middle molecular weight blood peptides].
Accumulation of biologically active peptides, distinctly affecting the respiration and enzymatic activity of mitochondria, may be responsible for deterioration of oxidative phosphorylation occurring in mitochondria within the early period of burn toxemia.
D000818:Animals; D001681:Biological Assay; D002056:Burns / Q000097:blood* / Q000150:complications / Q000378:metabolism; D004285:Dogs; D051379:Mice; D008930:Mitochondria, Liver / Q000201:enzymology / Q000378:metabolism*; D010084:Oxidation-Reduction; D010101:Oxygen Consumption; D010455:Peptides / Q000097:blood* / Q000494:pharmacology; D014115:Toxemia / Q000097:blood* / Q000150:complications / Q000378:metabolism; D014118:Toxins, Biological / Q000097:blood* / Q000494:pharmacology
D010455:Peptides; D014118:Toxins, Biological
Funktsional'noe sostoianie mitokhondriĭ v period ranneĭ ozhogovoĭ toxemii i pri vozdeĭstvii srednemolekuliarnykh peptidov krovi.