[Hypothesis on the basic immune disorders, the originating cause and the development of glomerulonephritis].
After a detailed survey on the latest achievements in the immunology of glomerulonephritis, the most important results from the author's studies in that field are presented. A new working hypothesis is proposed, on the base of the results and the literature data, for the origination and development of glomerulonephritis.
D000936:Antigen-Antibody Complex / Q000032:analysis; D001323:Autoantibodies / Q000032:analysis; D001327:Autoimmune Diseases / Q000209:etiology* / Q000276:immunology; D001485:Basement Membrane / Q000276:immunology; D003176:Complement C3 / Q000032:analysis; D005921:Glomerulonephritis / Q000209:etiology* / Q000276:immunology; D006801:Humans; D007070:Immunoglobulin A / Q000032:analysis; D007074:Immunoglobulin G / Q000032:analysis; D007075:Immunoglobulin M / Q000032:analysis; D007678:Kidney Glomerulus / Q000276:immunology; D008213:Lymphocyte Activation; D012397:Rosette Formation
D000936:Antigen-Antibody Complex; D001323:Autoantibodies; D003176:Complement C3; D007070:Immunoglobulin A; D007074:Immunoglobulin G; D007075:Immunoglobulin M
Khipoteza za osnovnite imunni narusheniia, prichina za vŭznikvane i razvitie na glomerulonefritite.