Pulsatile flow of non-Newtonian fluid in distensible models of human arteries.
In addition to biochemical factors, hydromechanical influences are responsible for atherogenesis and deposits of blood platelets at bends and bifurcations of human arteries. Hence the flow patterns were simulated in a true-to-scale three-dimensional bifurcation of a human renal artery model and of an arterial femoralis with Newtonian and non-Newtonian blood like fluid. Investigations were made with steady and pulsatile flow. The velocity profiles (at physiological Re-numbers) were measured after the bifurcations with a laser-Doppler-anemometer. In previous works Newtonian fluids were used to investigate the flow in bends and bifurcations of rigid and elastic simplified models. In this paper, emphasis is placed on the difference between rigid and elastic models and also Newtonian and non Newtonian flow behavior. Differences between Newtonian and non Newtonian fluids may especially be expected to occur after branches where the flow has local strong convective elements such as in reverse zones and flow separation points.
D001158:Arteries / Q000502:physiology*; D001161:Arteriosclerosis / Q000209:etiology*; D001783:Blood Flow Velocity; D001809:Blood Viscosity; D004548:Elasticity; D005263:Femoral Artery / Q000502:physiology; D006801:Humans; D008875:Middle Aged; D008955:Models, Cardiovascular; D012077:Renal Artery / Q000502:physiology; D012212:Rheology