[Hypophosphatemic rickets in premature infants independent of vitamin D].
The etiology and prevention of rickets in prematures infants are still controversial: insufficient storage or intake in minerals and/or vitamin D, immature vitamin D metabolism, necessitating early vitamin supplementation, for some associated with calcium and phosphate supplementation. The authors report a case of rickets which could be related to an immaturity of the tubular mechanisms of phosphate reabsorption, with a favourable outcome following an increase in calcium and phosphate intake.
D002118:Calcium / Q000627:therapeutic use; D006801:Humans; D007231:Infant, Newborn; D007235:Infant, Premature, Diseases / Q000503:physiopathology*; D007684:Kidney Tubules / Q000503:physiopathology*; D008297:Male; D010710:Phosphates / Q000097:blood* / Q000378:metabolism / Q000627:therapeutic use; D012279:Rickets / Q000209:etiology* / Q000503:physiopathology / Q000628:therapy; D013997:Time Factors
D010710:Phosphates; D002118:Calcium
Rachitisme hypophosphatémique du prématuré indépendant de la vitamine D.