[Lipid peroxidation in experimental myocardial infarct: the action of hyperbaric oxygenation].
Rats with experimental myocardial infarction demonstrated decrease in the activity of superoxide dismutase and catalase and increase in the content of lipid peroxidation (LPO) products and Schiff bases both in and outside the area of necrosis. The combined ischemic damage and hyperbaric oxygenation resulted in the over additive effect of accumulation of LPO products in and outside the area of infarction. The data suggest that it is desirable to use antioxidants during hyperbaric oxygenation.
D000818:Animals; D002374:Catalase / Q000378:metabolism; D006931:Hyperbaric Oxygenation; D008054:Lipid Peroxides / Q000378:metabolism*; D008297:Male; D009203:Myocardial Infarction / Q000378:metabolism* / Q000628:therapy; D009206:Myocardium / Q000378:metabolism; D010084:Oxidation-Reduction; D051381:Rats; D011919:Rats, Inbred Strains; D012545:Schiff Bases / Q000378:metabolism; D013482:Superoxide Dismutase / Q000378:metabolism
D008054:Lipid Peroxides; D012545:Schiff Bases; D002374:Catalase; D013482:Superoxide Dismutase
Perekisnoe okislenie lipidov pri éksperimental'nom infarkte miokarda: deĭstvie giperbaricheskoĭ oksigenatsii.