[Physiopathology of angina pectoris].
The concept of blood flow limited fixed coronary artery stenosis holds true for most of the patients with stable, e.g. exercise-dependent angina pectoris. In this condition exists a disproportion between oxygen demand and oxygen consumption. On the other hand the unstable angina pectoris (angina pectoris at rest) is explained by a variable transient reduction of oxygen supply due to vasoconstriction ("dynamic stenosis"). In addition to the fixed stenosis and vasoconstriction the rheological behaviour plays a further role in the pathogenesis.
D000787:Angina Pectoris / Q000503:physiopathology*; D000788:Angina Pectoris, Variant / Q000503:physiopathology; D003326:Coronary Circulation; D003327:Coronary Disease / Q000503:physiopathology; D003329:Coronary Vasospasm / Q000503:physiopathology; D004562:Electrocardiography; D006329:Heart Conduction System / Q000503:physiopathology; D006439:Hemodynamics; D006801:Humans; D009203:Myocardial Infarction / Q000503:physiopathology; D009206:Myocardium / Q000378:metabolism; D010101:Oxygen Consumption; D010974:Platelet Aggregation; D012212:Rheology
Pathophysiologie der Angina pectoris.