[Various aspects of the pathogenesis of the postischemic syndrome].
The contribution of some regulatory humoral systems (sympathoadrenal, pituitary-adrenal, kallikrein-kinin and renin-angiotensin ones) to the development of postischemic hemodynamic disorders associated with acute arterial occlusion are demonstrated. Pathogenetic rationale is offered for methods of postischemic cardiovascular functional disorders control (the use of antienzyme drugs, cortisol, tocopherol on days 1-3 of the postischemic period).
D000313:Adrenal Medulla / Q000503:physiopathology; D004950:Esterases / Q000097:blood; D005121:Extremities / Q000098:blood supply*; D006801:Humans; D007511:Ischemia / Q000503:physiopathology*; D007610:Kallikreins / Q000037:antagonists & inhibitors; D010913:Pituitary-Adrenal System / Q000503:physiopathology; D011288:Prekallikrein / Q000097:blood; D012084:Renin-Angiotensin System; D013564:Sympathetic Nervous System / Q000503:physiopathology; D013577:Syndrome
D011288:Prekallikrein; D004950:Esterases; D007610:Kallikreins
Nekotorye aspekty patogeneza postishemicheskogo sindroma.