Skeletal muscle: anatomy, physiology, and pathophysiology.
Many questions remain to be answered. The intention here is to attempt to place in perspective a portion of what is known and understood scientifically, to make working sense of the pathophysiologic process so that we as clinicians can "in our mind's eye" understand what it is that we are trying to treat with what now seems to be a multitude of therapies. When a patient presents to your office with acute or chronic, or both, myofascial pain dysfunction syndrome, some basic questions must be addressed. Is the disease process genetic (intrinsic) or acquired (extrinsic), or both? It is the purpose of diagnosis to unearth the underlying predispositions that patients may exhibit. Often the predispositions are quite obvious (gross postural discrepancies and skeletal and dental malrelationships( or they may be consummately subtle (endocrinopathies and behavioral patterns). It seems that the main job to be completed is diagnosis followed by the utilization of well-known physical medicine (conservative) techniques that treat the source of the disorder: the myofascial trigger zone.
D000255:Adenosine Triphosphate / Q000378:metabolism; D001696:Biomechanical Phenomena; D006801:Humans; D009119:Muscle Contraction; D009124:Muscle Proteins / Q000502:physiology; D009132:Muscles / Q000033:anatomy & histology / Q000378:metabolism / Q000502:physiology*; D013035:Spasm / Q000503:physiopathology; D013706:Temporomandibular Joint Dysfunction Syndrome / Q000503:physiopathology
D009124:Muscle Proteins; D000255:Adenosine Triphosphate