[Functional tricuspid regurgitation and its relation to the morphology of the tricuspid valve and annulus: pulsed doppler echocardiography and two-dimensional echocardiography].
The mechanism for the development of functional tricuspid regurgitation (TR) was studied by an ultrasonic method. Thirty-five examinations were performed in 31 patients who were expected to have functional TR, and the severity was classified into 4 grades according to the extension of the regurgitant signals by pulsed Doppler echocardiography. The satisfactory horizontal section of the tricuspid valve was obtained by two-dimensional echocardiography (2DE) to measure the tricuspid annular diameter and to observe systolic configuration of the tricuspid valve in 22 examinations. The tricuspid annular diameter was well correlated with the severity of TR, and "lack of coaptation" of the valve was recognized on 2DE in some cases of severe TR with the markedly dilated annulus, indicating that this dilatation was an important trigger of functional TR. Additionally, in the majority of patients with severe TR, "anterior displacement" of the tips of tricuspid leaflet(s) (6 mm or more from the tricuspid annulus towards the right ventricle) was observed, which was thought to be due to the chordal traction secondary to the right ventricular dilatation, and contributed to the development of functional TR by disturbing sufficient coaptation. In one particular case, severe TR was associated with " malaligned coaptation" caused by the anterior displacement confined to the septal leaflet, indicating that asymmetrical dilatation of the right ventricle and/or disorientation of chordae-valve system may contribute to TR.(ABSTRACT TRUNCATED AT 250 WORDS)
D004108:Dilatation, Pathologic; D004452:Echocardiography / Q000379:methods; D006801:Humans; D014261:Tricuspid Valve / Q000473:pathology*; D014262:Tricuspid Valve Insufficiency / Q000175:diagnosis / Q000209:etiology* / Q000503:physiopathology