[Etiopathogenetic considerations in 2 cases of dysmetabolic hypokalemic paralysis with signs of increased neuromuscular excitability during potassium repletion].
Increased neuromuscular excitability with varying clinical and EMG features were also observed during KCl administration in both cases. The findings are discussed on the light of the membrane ionic gradients current theory.
D000328:Adult; D005260:Female; D006801:Humans; D007008:Hypokalemia / Q000188:drug therapy / Q000209:etiology* / Q000503:physiopathology; D008297:Male; D008564:Membrane Potentials; D008875:Middle Aged; D009132:Muscles / Q000503:physiopathology; D009469:Neuromuscular Junction / Q000502:physiology*; D010243:Paralysis / Q000188:drug therapy / Q000209:etiology* / Q000503:physiopathology; D011189:Potassium Chloride / Q000627:therapeutic use
D011189:Potassium Chloride
Considerazioni eziopatogenetiche su due casi di paralisi ipokaliemica dismetabolica con manifestazioni di aumentata eccitabilità neuromuscolare durante replezione potassica.