Lysosomal hydrolases of dog kidney in superior mesenteric artery occlusion (SMAO) shock.
In 6 dogs with superior mesenteric artery occlusion (SMAO) shock and in 6 healthy animals the total and free activity of lysosomal hydrolases (acid phosphatase, betaglucuronidase and cathepsins) in the whole homogenate, the lysosomal enriched subfraction and the supernatant of kidneys were estimated. Statistically significant differences between the two groups were not observed. These results indicate that in SMAO shock, the stability of kidney lysosomes is well preserved during observed period.
D000135:Acid Phosphatase / Q000378:metabolism; D000818:Animals; D002403:Cathepsins / Q000378:metabolism; D004285:Dogs; D005260:Female; D005966:Glucuronidase / Q000378:metabolism; D006867:Hydrolases / Q000378:metabolism*; D007668:Kidney / Q000648:ultrastructure*; D008247:Lysosomes / Q000201:enzymology / Q000502:physiology*; D008297:Male; D008638:Mesenteric Arteries; D008641:Mesenteric Vascular Occlusion / Q000201:enzymology / Q000473:pathology*; D012769:Shock / Q000201:enzymology / Q000209:etiology / Q000473:pathology*
D006867:Hydrolases; D000135:Acid Phosphatase; D005966:Glucuronidase; D002403:Cathepsins