Hypercalcaemia induced by increased thyroxine substitution in a patient treated with dihydrotachysterol.
The metabolism of vitamin D is essential in the control of bone and mineral metabolism. Hyperthyroidism as well as hypothyroidism effect the metabolism of bone tissue and vitamin D. We present a dihydrotachysterol-calcium treated patient with post-operative hypothyroidism, who developed hypercalcaemia, when the thyroxine dosage was increased.
D002118:Calcium / Q000009:adverse effects; D004097:Dihydrotachysterol / Q000009:adverse effects*; D005260:Female; D006801:Humans; D006934:Hypercalcemia / Q000139:chemically induced*; D006996:Hypocalcemia / Q000188:drug therapy / Q000209:etiology; D007037:Hypothyroidism / Q000188:drug therapy / Q000209:etiology; D008875:Middle Aged; D011183:Postoperative Complications / Q000188:drug therapy; D013974:Thyroxine / Q000009:adverse effects*
D013974:Thyroxine; D004097:Dihydrotachysterol; D002118:Calcium