[Hutchinson-Horton giant cell arteritis and rhizomelic polymyalgia. III. Current pathogenetic and nosographic aspects].
There is much evidence to suggest that temporal arteritis and rhizomelic polymyalgia are both immunological diseases. The classic results of experimental pathology are discussed, together with the relations between rhizomelic polymyalgia and both virus hepatitis B and the HLA system. From the clinical standpoint, it is now agreed that differences in individual response may lead to either a synovial or an arteritic response in both forms. Their association in what Hamrin has called "polymyalgia arteritica" is also common.
D000936:Antigen-Antibody Complex / Q000276:immunology; D000956:Antigens, Viral / Q000276:immunology; D004549:Elastin / Q000276:immunology; D013700:Giant Cell Arteritis / Q000150:complications* / Q000276:immunology; D006680:HLA Antigens / Q000276:immunology; D006509:Hepatitis B / Q000150:complications; D006515:Hepatitis B virus / Q000276:immunology; D006801:Humans; D006968:Hypersensitivity, Delayed / Q000276:immunology; D011111:Polymyalgia Rheumatica / Q000209:etiology* / Q000276:immunology
D000936:Antigen-Antibody Complex; D000956:Antigens, Viral; D006680:HLA Antigens; D004549:Elastin
L'arterite gigantocellulare di Hutchinson-Horton e la polimialgia rizomelica. Nota III. Aspetti patogenetici e nosografici attuali.