title: [Artificial respiration in cerebral resuscitation].
abstract: CO2 is a potent cerebral vasodilator which induces major changes in cerebral blood flow and volume. Acute hypocapnia decreases cerebral blood volume; thus the intracranial pressure can be reduced rapidly, thereby improving cerebral hemodynamics. Hypocapnia, rapidly achieved by controlled hyperventilation, is therefore a valid treatment in cerebral resuscitation, especially when associated with elevated intracranial pressure. However, the effect of hypocapnia is short and unusually does not exceed a few hours. Consequently the indications for prolonged hyperventilation are very limited, if they exist at all. Intracranial pressure and cerebral blood flow changes induced by PEEP are still controversial.
mesh_terms: D002245:Carbon Dioxide / Q000097:blood*; D002560:Cerebrovascular Circulation; D006801:Humans; D006985:Hyperventilation / Q000503:physiopathology*; D007427:Intracranial Pressure; D012121:Respiration, Artificial; D013997:Time Factors
chemical_list: D002245:Carbon Dioxide
vernacular_title: Ventilation artificielle en réanimation cérébrale.