Complement elevation in spinal cord injury.
Laboratory studies revealed an elevated complement in 66% of patients with spinal cord injury. It is postulated that the activated complement may be a component of self-feeding immunological mechanism responsible for the failure of regeneration of a mature mammalian spinal cord. There was no evidence that such an injury had any effect on pre-existing atopy.
D000293:Adolescent; D000328:Adult; D000974:Antibodies, Antinuclear; D011415:Complement Factor B; D003165:Complement System Proteins; D005260:Female; D006801:Humans; D006969:Hypersensitivity, Immediate / Q000276:immunology; D007070:Immunoglobulin A; D007073:Immunoglobulin E; D007074:Immunoglobulin G; D007075:Immunoglobulin M; D008297:Male; D013119:Spinal Cord Injuries / Q000276:immunology*
D000974:Antibodies, Antinuclear; D007070:Immunoglobulin A; D007074:Immunoglobulin G; D007075:Immunoglobulin M; D007073:Immunoglobulin E; D003165:Complement System Proteins; D011415:Complement Factor B