[Neurophysiology of the pain syndrome in lumbar osteochondrosis].
Data on the character of evoked central responses of the soleus and the anterior tibial muscles, as well as the minor muscles of the foot sole and patients with the so-called discogenic lumbar radiculitis are presented. In addition, results of treating this pathology by the method of anti-algetic supracutaneous electric stimulation are described. On the basis of comparing all the data it is concluded that of importance in the neurophysiological mechanism of the pain in lumbar osteochondrosis are not only hyperimpulsation alone the thin pain-conducting fibres, but also insufficient conduction of signals alone large-diameter afferent fibres, as well as weakening of the segmental inhibitory processes and deterioration of the functioning of the antinociceptive system.
D000698:Analgesia; D004599:Electric Stimulation Therapy; D004594:Electrophysiology; D006801:Humans; D007405:Intervertebral Disc Displacement / Q000503:physiopathology*; D008159:Lumbar Vertebrae; D009408:Nerve Compression Syndromes / Q000503:physiopathology; D010007:Osteochondritis / Q000503:physiopathology*; D010146:Pain / Q000503:physiopathology*; D010525:Peripheral Nerves / Q000503:physiopathology*; D013122:Spinal Diseases / Q000503:physiopathology; D013577:Syndrome
Neĭrofiziologiia bolevogo sindroma pri poiasnichnom osteokhondroze.