[Alcohol and lipid metabolism].
Alcohol has a direct and/or indirect influence on the fat metabolism, evoking a HLP and this again may lead to secondary defects. Liver defects, pancreatitis, haemolytic conditions may be the sequel of the alcohol as well as of the HLP. When a HLP, particularly of type IV. is present, these statements demand a careful alcohol anamnesis. A control after absolute alcohol abstinence is necessary. If within a few days the HLP shows tendencies to involution, the alcohol can be proved as pathogenetic factor. The exclusion of the noxa is of decisive therapeutic importance and often spares a little effective and loaded with side-effects pharmacotherapy.
D000429:Alcohol Oxidoreductases / Q000378:metabolism; D000437:Alcoholism / Q000150:complications / Q000378:metabolism; D000743:Anemia, Hemolytic / Q000150:complications; D000431:Ethanol / Q000378:metabolism*; D005230:Fatty Acids, Nonesterified / Q000097:blood; D006801:Humans; D006951:Hyperlipoproteinemias / Q000209:etiology; D007565:Jaundice / Q000209:etiology; D050356:Lipid Metabolism; D008099:Liver / Q000201:enzymology*; D009243:NAD / Q000378:metabolism; D010195:Pancreatitis / Q000150:complications; D013577:Syndrome; D014527:Uric Acid / Q000097:blood
D005230:Fatty Acids, Nonesterified; D009243:NAD; D014527:Uric Acid; D000431:Ethanol; D000429:Alcohol Oxidoreductases
Alkohol und Fettstoffwechsel.