[Effect of ethanolamine on corticosterone concentration in experimental myocardial hyperfunction in the white rat].
An initial step of experimental myocardium hyperfunction, caused by coarctation of the subphrenic part of abdominal aorta, was accompanied by a distinct increase in content of corticosterone in rat blood and adrenal glands. Administration of ethanolamine at a dose of 10 mg/kg decreased the hormone content under conditions of heart hyperfunction. The data obtained suggest that adrenal gland cortex was activated in response to the myocardium pathology as well as that content of corticosterone in blood and adrenal glands might be regulated by administration of ethanolamine.
D000302:Adrenal Cortex / Q000503:physiopathology*; D000311:Adrenal Glands / Q000032:analysis; D000818:Animals; D001017:Aortic Coarctation / Q000097:blood*; D003345:Corticosterone / Q000032:analysis* / Q000097:blood; D019856:Ethanolamine; D004983:Ethanolamines; D006321:Heart / Q000503:physiopathology*; D008297:Male; D010913:Pituitary-Adrenal System / Q000503:physiopathology; D051381:Rats
D004983:Ethanolamines; D019856:Ethanolamine; D003345:Corticosterone
Deĭstvie étanolamina na soderzhanie kortikosterona u belykh krys s éksperimental'noĭ giperfunktsieĭ miokarda.