Further studies on the role of calcium in the depolarization-induced activation of tryptophan hydroxylase. Effect of verapamil, Tetracaine, haloperidol and fluphenazine.
D000818:Animals; D001933:Brain Stem / Q000201:enzymology*; D002021:Buffers / Q000494:pharmacology; D002118:Calcium / Q000502:physiology*; D004789:Enzyme Activation / Q000187:drug effects; D005476:Fluphenazine / Q000494:pharmacology; D006220:Haloperidol / Q000494:pharmacology; D008297:Male; D008564:Membrane Potentials / Q000187:drug effects; D011188:Potassium / Q000494:pharmacology; D051381:Rats; D011919:Rats, Inbred Strains; D013748:Tetracaine / Q000494:pharmacology; D014365:Tryptophan Hydroxylase / Q000378:metabolism*; D014700:Verapamil / Q000494:pharmacology
D002021:Buffers; D013748:Tetracaine; D014700:Verapamil; D014365:Tryptophan Hydroxylase; D006220:Haloperidol; D011188:Potassium; D005476:Fluphenazine; D002118:Calcium