[Detrusor inactivity in central spinal cord lesions. Hypothesis of inhibitory hyperreflexia of sympathetic origin].
In connection with two observations of suprasacral complete spinal cord patients showing a clear sublesional spasticity and paradoxically and inactive and unreactive detrusor, the authors discuss the mechanisms that are usually considered: vesical distension, sacral peripheric neurogenic lesion, reflex inhibition of the detrusor of somatic origin. They express the hypothesis of an inhibition hyper-reflex of sympathetic origin, reversible under phenoxybenzamine treatment, consider the urodynamic evaluation and the therapeutic treatment and compare this type of neurogenic vesical dysfunction to that of the spinal shock.
D000293:Adolescent; D000328:Adult; D006801:Humans; D008297:Male; D009433:Neural Inhibition; D012021:Reflex, Abnormal / Q000209:etiology*; D013119:Spinal Cord Injuries / Q000150:complications*; D013564:Sympathetic Nervous System / Q000503:physiopathology*; D001743:Urinary Bladder / Q000294:innervation; D001750:Urinary Bladder, Neurogenic / Q000209:etiology* / Q000503:physiopathology / Q000628:therapy; D014555:Urination Disorders / Q000209:etiology; D014563:Urodynamics
Inactivité du détrusor dans les lésions médullaires centrales. Hypothèse d'une hyperréflexie d'inhibition d'origine sympathique.