[Control of lipid metabolism in experimental obesity].
Experimental obesity syndromes are characterized by considerable fat accretion which may be the result of hyperinsulinemia. Adipose tissue accretion may be the result both of the excessive synthesis of triglycerides and a defect in their mobilization from adipose tissue stores. The adenylate cyclase system which appears to be the site of the defect in mobilization is studies in more detail.
D000262:Adenylyl Cyclases / Q000378:metabolism; D000273:Adipose Tissue / Q000378:metabolism*; D000818:Animals; D002462:Cell Membrane / Q000378:metabolism; D004195:Disease Models, Animal; D006160:Guanosine Triphosphate / Q000494:pharmacology; D006165:Guanylyl Imidodiphosphate / Q000494:pharmacology; D007700:Kinetics; D008066:Lipolysis; D008099:Liver / Q000378:metabolism*; D051379:Mice; D008820:Mice, Obese; D009765:Obesity / Q000235:genetics / Q000378:metabolism*; D051381:Rats; D011919:Rats, Inbred Strains; D011924:Rats, Zucker
D006165:Guanylyl Imidodiphosphate; D006160:Guanosine Triphosphate; D000262:Adenylyl Cyclases
Contrôle du métabolisme lipidique dans l'obésité expérimentale.