[Pathophysiology of tetanus rigidity (author's transl)].
Brainstem reflexes have been studied electrophysiologically in six patients with generalized tetanus. The selective impairment of reflexes with long polysynaptic pathways supports the assumption of an interneuronal dysfunction in tetanus. The behaviour of proprioceptive reflexes suggests that hyperactivity of gamma-motoneurons contributes to the rigidity in an early stage of the disease.
D000328:Adult; D001921:Brain / Q000503:physiopathology*; D005260:Female; D006801:Humans; D008297:Male; D008875:Middle Aged; D009046:Motor Neurons / Q000187:drug effects; D012018:Reflex / Q000187:drug effects; D013742:Tetanus / Q000503:physiopathology*; D013744:Tetanus Toxin / Q000494:pharmacology
D013744:Tetanus Toxin
La physiopathologie de l'hypertonie musculaire du tétanus.