[Toxic nephropathies (author's transl)].
Direct tubular damage, hypersensitivity reaction, metabolically mediated kidney disturbances, and chronic nephropathies are important sequelae of several drugs or their metabolites. In this review the drug-induced kidney disease is discussed from a clinical, histological, and pathogenetic point of view. The knowledge of possible nephrotoxic reactions and their underlying toxins are essential for prevention of this kidney disease.
D000700:Analgesics / Q000009:adverse effects; D004342:Drug Hypersensitivity / Q000276:immunology; D005026:Ethylene Glycols / Q000009:adverse effects; D005921:Glomerulonephritis / Q000139:chemically induced; D006801:Humans; D006934:Hypercalcemia / Q000150:complications; D007671:Kidney Concentrating Ability; D007674:Kidney Diseases / Q000139:chemically induced*; D007676:Kidney Failure, Chronic / Q000139:chemically induced; D007683:Kidney Tubular Necrosis, Acute / Q000139:chemically induced; D008712:Methicillin / Q000009:adverse effects; D008727:Methotrexate / Q000009:adverse effects; D009395:Nephritis, Interstitial / Q000139:chemically induced; D010070:Oxalates / Q000009:adverse effects
D000700:Analgesics; D005026:Ethylene Glycols; D010070:Oxalates; D008712:Methicillin; D008727:Methotrexate
Toxische Nierenschäden.