Gap junction formation in myometrium: control by estrogens, progesterone, and prostaglandins.
D000818:Animals; D001095:Arachidonic Acids / Q000494:pharmacology; D004537:Eicosanoic Acids / Q000494:pharmacology; D004967:Estrogens / Q000494:pharmacology*; D005260:Female; D005614:Freeze Fracturing; D066298:In Vitro Techniques; D007213:Indomethacin / Q000494:pharmacology; D007365:Intercellular Junctions / Q000648:ultrastructure*; D008854:Microscopy, Electron; D009215:Myometrium / Q000648:ultrastructure*; D011247:Pregnancy; D011374:Progesterone / Q000494:pharmacology; D011448:Prostaglandin Antagonists / Q000494:pharmacology*; D011453:Prostaglandins / Q000494:pharmacology*; D011458:Prostaglandins E / Q000494:pharmacology; D011460:Prostaglandins F / Q000494:pharmacology; D051381:Rats; D013929:Thromboxane B2 / Q000494:pharmacology; D014599:Uterus / Q000648:ultrastructure*
D001095:Arachidonic Acids; D004537:Eicosanoic Acids; D004967:Estrogens; D011448:Prostaglandin Antagonists; D011453:Prostaglandins; D011458:Prostaglandins E; D011460:Prostaglandins F; D011374:Progesterone; D013929:Thromboxane B2; D007213:Indomethacin