[Long-term observation of pulmonary water and protein extravasation in traumatic-hemorrhagic shock (author's transl)].
Eleven mongrel dogs were observed for 72 h following a standardized traumatic hemorrhagic shock. The decrease of plasma albumin concentration and blood 125-I-albumin activity with a concomitant increase in lung I-albumin content is possibly caused by capillary leakage. A rise in the extravascular lung water content was measured by a gravimetric postmortem method. An in vivo double indicator technique, however, failed to document these findings.
D000818:Animals; D001798:Blood Proteins / Q000378:metabolism; D002199:Capillary Permeability; D004285:Dogs; D005110:Extracellular Space / Q000378:metabolism; D012128:Respiratory Distress Syndrome / Q000378:metabolism*; D012771:Shock, Hemorrhagic / Q000378:metabolism*; D012774:Shock, Traumatic / Q000378:metabolism*
D001798:Blood Proteins
Verlauf des pulmonalen Wasser- und Eiweissaustritts im protrahierten traumatisch-hämorrhagischen Schock.