The kidney in health and disease: XVIII: lactic acidosis and the elevated anion gap (II).
This concluding article of a two-part discussion deals with clinical presentations and management of the basic types of lactic acidosis - with and without appaent hypoxia - and the various complicating conditions that may coexist with them. Although symptomatic treatment is essential, cure can be effected only by seeking and treating the underlying cause of the electrolyte derangement.
D000138:Acidosis / Q000175:diagnosis* / Q000188:drug therapy / Q000378:metabolism; D001639:Bicarbonates / Q000627:therapeutic use; D001774:Blood Chemical Analysis; D003920:Diabetes Mellitus / Q000378:metabolism; D006439:Hemodynamics; D006801:Humans; D006863:Hydrogen-Ion Concentration; D000860:Hypoxia / Q000378:metabolism; D007773:Lactates / Q000097:blood*; D009369:Neoplasms / Q000378:metabolism; D010629:Phenformin / Q000009:adverse effects; D011768:Pyruvate Dehydrogenase Complex / Q000097:blood; D014883:Water-Electrolyte Imbalance / Q000378:metabolism
D001639:Bicarbonates; D007773:Lactates; D011768:Pyruvate Dehydrogenase Complex; D010629:Phenformin