[Effect of acute stress on structuro-functional parameters of mouse liver and adrenal cortex].
The acute stress following nervous and physical loading in connection with abnormally high functional demands to the liver, may be an independent etiological factor of development of hepatocytes destruction process. These phenomena are based on catabolic direction of metabolism in hepatocytes of animals under stress at the second neuroendocrine phase of the stress. At the same time the results of the investigations show the possibility of forming the adaptive reactions at the ultrastructural level in response to the acute stress.
D015062:11-Hydroxycorticosteroids / Q000378:metabolism; D000208:Acute Disease; D000302:Adrenal Cortex / Q000378:metabolism*; D000305:Adrenal Cortex Hormones / Q000378:metabolism*; D000818:Animals; D002466:Cell Nucleolus / Q000648:ultrastructure; D002467:Cell Nucleus / Q000648:ultrastructure; D002843:Chromatin / Q000648:ultrastructure; D003345:Corticosterone / Q000378:metabolism; D008099:Liver / Q000378:metabolism / Q000648:ultrastructure*; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D013312:Stress, Physiological / Q000473:pathology*
D015062:11-Hydroxycorticosteroids; D000305:Adrenal Cortex Hormones; D002843:Chromatin; D003345:Corticosterone
Vliianie ostrogo stressa na strukturno-funktsional'nye pokazateli pecheni i kory nadpochechnikov mysheĭ.