[Behavior of thyroid hormones, their regulation and relations to the clinical course of myocardial infarction].
With the help of a clinical study during the acute phase of the myocardial infarction the behaviour of the triiodothyronine (T3) in correlation to the creatinine phosphokinase (CPK) and clinical parameters was examined. The CPK and T3 exhibit a reciprocal behaviour. In dependence on the clinical stage of the acute myocardial infarction three reaction patterns of the T3 behaviour are described and discussed. It is referred to the relation of the neuro-hormonal regulation mechanism to the changed metabolic behaviour in the acute myocardial infarction. Possibilities of the importance of T3 and its peripheral metabolism in the metabolic regulation are shown.
D000328:Adult; D003402:Creatine Kinase / Q000097:blood; D005260:Female; D006801:Humans; D008297:Male; D008875:Middle Aged; D009203:Myocardial Infarction / Q000097:blood* / Q000201:enzymology; D011863:Radioimmunoassay; D014284:Triiodothyronine / Q000097:blood*
D014284:Triiodothyronine; D003402:Creatine Kinase
Das Verhalten der Schilddrüsenhormone, ihre Regulation und Beziehungen zum klinischen Verlauf des Myokardinfarktes.