Dissociation of delayed-type hypersensitivity from acquired murine resistance to Brucella suis.
Mouse pretreatment with cyclophosphamide or acetoacetylation of the vaccinating fraction "PI" lead to a dissociation between enhanced anti-PI DTH responses and normal or depressed acquired resistance to an infective challenge with Brucella suis.
D000107:Acetylation; D000818:Animals; D000907:Antibodies, Bacterial / Q000096:biosynthesis; D002002:Brucella / Q000276:immunology*; D005591:Chemical Fractionation; D003520:Cyclophosphamide / Q000494:pharmacology; D005260:Female; D006968:Hypersensitivity, Delayed / Q000276:immunology*; D007111:Immunity, Cellular; D051379:Mice; D014611:Vaccination
D000907:Antibodies, Bacterial; D003520:Cyclophosphamide