[Neurochemical mechanisms of caudate nucleus participation in the performance of food-getting behavior].
The study was concerned with the significance of acetylcholine (ACh)- and dophaminergic (DA) systems of the caudate nucleus in the control of food-procuring behaviour in cats. Neurochemical stimulation of ACh-ergic structures of the caudate nucleus inhibited primarily the motor component of the conditioned food-procuring reflex. Neurochemical action on DA-ergic structures had a considerably less pronounced influence on this reflex. The analysis of changes in the amplitude of evoked potentials in the motor and somatosensory cortical areas to conditioned sound stimuli after microinjections in the caudate nucleus of ACh- and DA-trophic drugs did not reveal any antagonism between ACh- and DA-ergic systems of the caudate nucleus.
D000109:Acetylcholine / Q000502:physiology*; D000818:Animals; D001058:Apomorphine / Q000494:pharmacology; D001285:Atropine / Q000494:pharmacology; D001931:Brain Mapping; D002217:Carbachol / Q000494:pharmacology; D002415:Cats; D002421:Caudate Nucleus / Q000187:drug effects / Q000502:physiology*; D003214:Conditioning, Classical / Q000502:physiology*; D004298:Dopamine / Q000494:pharmacology / Q000502:physiology*; D005071:Evoked Potentials; D005247:Feeding Behavior / Q000502:physiology*; D006220:Haloperidol / Q000494:pharmacology; D009388:Neostigmine / Q000494:pharmacology; D013016:Sound
D009388:Neostigmine; D001285:Atropine; D002217:Carbachol; D006220:Haloperidol; D001058:Apomorphine; D000109:Acetylcholine; D004298:Dopamine
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