Suppression of experimental glomerulonephritis by interference with T- or B-cell activation signals or adhesion molecules.
Andres
G
G
Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston, USA.
Nishikawa
K
K
Stamenkovic
I
I
McCluskey
R T
RT
D000069594:Abatacept; D000818:Animals; D015703:Antigens, CD; D000943:Antigens, Differentiation / Q000502:physiology; D001402:B-Lymphocytes / Q000276:immunology*; D018122:B7-1 Antigen / Q000502:physiology; D018106:CD28 Antigens / Q000502:physiology; D060908:CTLA-4 Antigen; D015815:Cell Adhesion Molecules / Q000502:physiology*; D005921:Glomerulonephritis / Q000628:therapy*; D006801:Humans; D018796:Immunoconjugates; D008213:Lymphocyte Activation; D013601:T-Lymphocytes / Q000276:immunology*
D015703:Antigens, CD; D000943:Antigens, Differentiation; D018122:B7-1 Antigen; D018106:CD28 Antigens; D060908:CTLA-4 Antigen; C556706:CTLA4 protein, human; D015815:Cell Adhesion Molecules; D018796:Immunoconjugates; D000069594:Abatacept