Restriction of hypoxic membrane defect by glycine improves mitochondrial and cellular function in reoxygenated renal tubules.
Gronow
G
G
Department of Physiology, University of Kiel, Germany.
Klause
N
N
Mályusz
M
M
D000118:Acetylglucosaminidase / Q000378:metabolism; D000818:Animals; D005969:Glutamate Dehydrogenase / Q000378:metabolism; D005998:Glycine / Q000494:pharmacology*; D000860:Hypoxia / Q000188:drug therapy* / Q000378:metabolism* / Q000473:pathology; D066298:In Vitro Techniques; D007672:Kidney Cortex / Q000187:drug effects / Q000378:metabolism / Q000473:pathology; D007684:Kidney Tubules / Q000187:drug effects* / Q000378:metabolism* / Q000473:pathology; D007770:L-Lactate Dehydrogenase / Q000378:metabolism; D007773:Lactates / Q000378:metabolism; D019344:Lactic Acid; D008928:Mitochondria / Q000187:drug effects / Q000378:metabolism; D010101:Oxygen Consumption / Q000187:drug effects; D011188:Potassium / Q000378:metabolism; D051381:Rats
D007773:Lactates; D019344:Lactic Acid; D007770:L-Lactate Dehydrogenase; D005969:Glutamate Dehydrogenase; D000118:Acetylglucosaminidase; D011188:Potassium; D005998:Glycine