Serial positron emission tomography with fludeoxyglucose F 18 in Creutzfeldt-Jakob disease.
We serially examined a patient with Creutzfeldt-Jakob disease using fludeoxyglucose F 18 and positron emission tomography. Marked cerebral hypometabolism reflected clinical deterioration in the early stages of Creutzfeldt-Jakob disease when no parenchymal abnormalities were present on MR imaging.
Ogawa
T
T
Department of Radiology and Nuclear Medicine, Research Institute of Brain and Blood Vessels-Akita, Japan.
Inugami
A
A
Fujita
H
H
Hatazawa
J
J
Shimosegawa
E
E
Kanno
I
I
Okudera
T
T
Uemura
K
K
Nagata
K
K
D001786:Blood Glucose / Q000378:metabolism*; D001921:Brain / Q000000981:diagnostic imaging / Q000473:pathology; D007562:Creutzfeldt-Jakob Syndrome / Q000000981:diagnostic imaging* / Q000473:pathology; D003847:Deoxyglucose / Q000031:analogs & derivatives / Q000378:metabolism; D003937:Diagnosis, Differential; D004292:Dominance, Cerebral / Q000502:physiology; D004734:Energy Metabolism / Q000502:physiology; D017809:Fatal Outcome; D005260:Female; D019788:Fluorodeoxyglucose F18; D006801:Humans; D008875:Middle Aged; D014055:Tomography, Emission-Computed / Q000379:methods*
D001786:Blood Glucose; D019788:Fluorodeoxyglucose F18; D003847:Deoxyglucose