Does energy demand have an additional control in ischemia or are current models of metabolic control adequate at extremes?
Connett
R J
RJ
Department of Physiology, University of Rochester Medical Center, NY 14642, USA.
Gayeski
T E
TE
Honig
C R
CR
D000255:Adenosine Triphosphate / Q000378:metabolism; D000818:Animals; D003600:Cytosol / Q000378:metabolism; D004285:Dogs; D004734:Energy Metabolism / Q000502:physiology*; D006019:Glycolysis; D006454:Hemoglobins / Q000378:metabolism; D007511:Ischemia / Q000378:metabolism*; D008931:Mitochondria, Muscle / Q000378:metabolism; D008954:Models, Biological; D018482:Muscle, Skeletal / Q000098:blood supply / Q000378:metabolism; D009211:Myoglobin / Q000378:metabolism; D010084:Oxidation-Reduction; D010101:Oxygen Consumption / Q000502:physiology; D010725:Phosphocreatine / Q000378:metabolism
D006454:Hemoglobins; D009211:Myoglobin; D010725:Phosphocreatine; D000255:Adenosine Triphosphate