Reduced neonatal liver iron concentrations after uteroplacental insufficiency.
Neonatal liver (storage) but not heart (nonstorage) tissue iron concentrations were reduced by 60% at autopsy in 15 newborn infants who had gestations complicated by uteroplacental insufficiency because of maternal hypertension or Potter syndrome. The hepatic iron reductions in term and preterm infants, and with either antecedent condition, were similar.
Georgieff
M K
MK
Department of Pediatrics at the University of Minnesota School of Medicine, Minneapolis, USA.
MIlls
M M
MM
Gordon
K
K
Wobken
J D
JD
D016022:Case-Control Studies; D005260:Female; D006801:Humans; D006973:Hypertension / Q000150:complications; D007231:Infant, Newborn / Q000378:metabolism; D007234:Infant, Premature / Q000378:metabolism; D007236:Infant, Small for Gestational Age / Q000378:metabolism; D007501:Iron / Q000032:analysis*; D000090463:Iron Deficiencies; D008099:Liver / Q000737:chemistry* / Q000473:pathology; D009206:Myocardium / Q000737:chemistry / Q000473:pathology; D010927:Placental Insufficiency / Q000150:complications*; D011247:Pregnancy; D011249:Pregnancy Complications, Cardiovascular
D007501:Iron