Amyloid beta protein disruption of cholinergic and growth factor phospholipase C signals could underlie cognitive and neurodegerative aspects of Alzheimer's disease.
Crews
F T
FT
Department of Pharmacology, University of Florida, Gainesville 32610-0267.
D000544:Alzheimer Disease / Q000201:enzymology / Q000378:metabolism* / Q000473:pathology*; D016229:Amyloid beta-Peptides / Q000502:physiology*; D003071:Cognition / Q000502:physiology; D006801:Humans; D009410:Nerve Degeneration / Q000502:physiology; D009414:Nerve Growth Factors / Q000502:physiology*; D010275:Parasympathetic Nervous System / Q000473:pathology*; D015398:Signal Transduction / Q000502:physiology; D010738:Type C Phospholipases / Q000502:physiology*
D016229:Amyloid beta-Peptides; D009414:Nerve Growth Factors; D010738:Type C Phospholipases