Pentoxifylline interferes with potential sources of free radical generation during endotoxemia.
Lerner
M R
MR
Department of Surgery, University of Oklahoma Health Sciences Center, Oklahoma City, USA.
Balla
A K
AK
Wilson
M F
MF
Brackett
D J
DJ
D000818:Animals; D001786:Blood Glucose / Q000378:metabolism; D002302:Cardiac Output / Q000187:drug effects; D004731:Endotoxins; D005609:Free Radicals / Q000378:metabolism; D006339:Heart Rate / Q000187:drug effects; D006439:Hemodynamics / Q000187:drug effects*; D006470:Hemorrhage / Q000517:prevention & control; D007003:Hypoglycemia; D007410:Intestinal Diseases / Q000517:prevention & control; D007773:Lactates / Q000097:blood; D008297:Male; D009504:Neutrophils / Q000187:drug effects / Q000502:physiology; D010431:Pentoxifylline / Q000494:pharmacology*; D051381:Rats; D017207:Rats, Sprague-Dawley; D012772:Shock, Septic / Q000378:metabolism / Q000503:physiopathology* / Q000517:prevention & control; D013318:Stroke Volume / Q000187:drug effects; D013997:Time Factors
D001786:Blood Glucose; D004731:Endotoxins; D005609:Free Radicals; D007773:Lactates; D010431:Pentoxifylline