Modulation of inflammatory activation pathways in the Lewis-to-F-344 rat chronic cardiac rejection model.
Russell
M E
ME
Department of Pathology, Harvard Medical School, Boston, Massachusetts 02115, USA.
Hancock
W W
WW
Wallace
A F
AF
Wyner
L R
LR
Karnovsky
M J
MJ
D000818:Animals; D001483:Base Sequence; D002908:Chronic Disease; D003324:Coronary Artery Disease / Q000276:immunology / Q000378:metabolism / Q000473:pathology; D016207:Cytokines / Q000502:physiology*; D017931:DNA Primers / Q000737:chemistry; D004041:Dietary Fats; D004195:Disease Models, Animal; D005228:Fatty Acids, Essential; D015870:Gene Expression; D006083:Graft Occlusion, Vascular / Q000378:metabolism / Q000517:prevention & control*; D006084:Graft Rejection; D016027:Heart Transplantation / Q000276:immunology*; D008969:Molecular Sequence Data; D009206:Myocardium / Q000378:metabolism; D012333:RNA, Messenger / Q000235:genetics; D051381:Rats; D011916:Rats, Inbred F344; D011917:Rats, Inbred Lew; D013154:Spleen / Q000378:metabolism
D016207:Cytokines; D017931:DNA Primers; D004041:Dietary Fats; D005228:Fatty Acids, Essential; D012333:RNA, Messenger