Early ischemic injury leads to long-term functional and morphologic deterioration of naive rat kidneys and may contribute to changes of chronic allograft rejection.
Tullius
S G
SG
Surgical Research Lab, Harvard Medical School, Boston, Massachusetts.
Heemann
U
U
Azuma
H
H
Pradham
P
P
Tilney
N L
NL
D000818:Animals; D015815:Cell Adhesion Molecules / Q000032:analysis; D002908:Chronic Disease; D006084:Graft Rejection / Q000473:pathology*; D000949:Histocompatibility Antigens Class II / Q000032:analysis; D018799:Intercellular Adhesion Molecule-1; D007511:Ischemia; D016030:Kidney Transplantation / Q000276:immunology / Q000473:pathology* / Q000502:physiology*; D008264:Macrophages / Q000276:immunology / Q000473:pathology; D009392:Nephrectomy; D009926:Organ Preservation; D051381:Rats; D011916:Rats, Inbred F344; D011917:Rats, Inbred Lew; D013601:T-Lymphocytes / Q000276:immunology / Q000473:pathology; D014184:Transplantation, Homologous
D015815:Cell Adhesion Molecules; D000949:Histocompatibility Antigens Class II; D018799:Intercellular Adhesion Molecule-1