[Several factors in the pathogenesis of hepatic failure in patients with liver cirrhosis and bleeding from esophageal and gastric varicose veins].
Bleeding in patients with liver cirrhosis leads to the activation of catabolic processes, accumulation of end products of nitrogen metabolism and pronounced imbalance of blood plasma amino acids. There is a correlation between the changes observed and disorders in neurological status. The authors hold that increased catabolism, pronounced imbalance of blood plasma amino acids and elevated blood ammonia level are the leading factors affecting the onset of hepatic encephalopathy.
Bour
A V
AV
Manuk'ian
G V
GV
Umruzakov
I U
IU
Shertsinger
A G
AG
D000596:Amino Acids / Q000097:blood / Q000378:metabolism; D000641:Ammonia / Q000097:blood; D004932:Esophageal and Gastric Varices / Q000097:blood / Q000150:complications* / Q000378:metabolism; D006471:Gastrointestinal Hemorrhage / Q000097:blood / Q000209:etiology* / Q000378:metabolism; D006501:Hepatic Encephalopathy / Q000097:blood / Q000209:etiology*; D006801:Humans; D008103:Liver Cirrhosis / Q000097:blood / Q000150:complications* / Q000378:metabolism; D009584:Nitrogen / Q000378:metabolism
D000596:Amino Acids; D000641:Ammonia; D009584:Nitrogen
Izuchenie nekotorykh faktorov patogeneza pechenochnoĭ nedostatochnosti u bol'nykh tsirrozom pecheni i krovotecheniem iz varikozno-rasshirennykh ven pishchevoda i zheludka.