[Hepatorenal insufficiency syndrome in endotoxic shock (a light optical and electron microscopic study)].
Experimental endotoxemia in the liver and kidneys was characterized by initial hemodynamic and rheologic disorders as well as intravascular coagulation disturbances some hours later. Hypoperfusion of these organs resulted in dystrophic changes in hepatocytes. Progression of endotoxin shock was accompanied by irreversible cellular changes. Late endotoxemia was characterized by adaptive changes associated with the improvement of microcirculation.
Kharlanova
N G
NG
Lomov
Iu M
IuM
Bardakhch'ian
E A
EA
D000818:Animals; D004285:Dogs; D005260:Female; D006439:Hemodynamics; D006530:Hepatorenal Syndrome / Q000209:etiology* / Q000473:pathology / Q000503:physiopathology; D007668:Kidney / Q000648:ultrastructure; D008099:Liver / Q000648:ultrastructure; D008297:Male; D012772:Shock, Septic / Q000150:complications* / Q000473:pathology / Q000503:physiopathology
Sindrom pechenochno-pochechnoĭ nedostatochnosti pri éndotoksinovom shoke (svetoopticheskoe i élektronno-mikroskopicheskoe issledovanie).