[Acute noninflammatory renal failure. New pathogenetic aspects in light of findings obtained in autopsy and biopsy of kidneys].
Thorough studies of kidney specimens obtained at autopsy or by renal biopsy revealed, that in noninflammatory acute renal failure oliguria/anuria is caused by increased activation o the renal renin-angiotensin system and increased generation of adenosine. At the polyuric phase moderately increased adenosine in kidney tissue seems to be the main factor responsible for decreased glomerular filtration.
Bohle
A
A
Instytut Patologii Uniwersytetu w Tybindze, Niemcy.
Kokot
F
F
Osswald
H
H
Schubert
B
B
Wehrmann
M
M
Erley
C
C
Risler
T
T
D058186:Acute Kidney Injury / Q000209:etiology / Q000473:pathology* / Q000503:physiopathology; D000241:Adenosine / Q000032:analysis; D000818:Animals; D001706:Biopsy; D006801:Humans; D007668:Kidney / Q000737:chemistry; D008297:Male; D009846:Oliguria / Q000209:etiology; D011141:Polyuria / Q000503:physiopathology; D012084:Renin-Angiotensin System / Q000502:physiology
D000241:Adenosine
Ostra niezapalna niewydolność nerek. Nowe aspekty patogenetyczne w świetle badań autopsyjnych i bioptatów nerkowych.