Dysfunctional recovery of prostacyclin synthase in aspirin-treated vascular cells: implications for prophylaxis of cardiovascular disease.
Dept. of Biochemistry and Molecular Biology, George Washington Medical School, Washington, D.C.
J M
JM
Bailey
K
K
Salata
A N
AN
Makheja
D016718:Arachidonic Acid / Q000378:metabolism; D001241:Aspirin / Q000494:pharmacology*; D002318:Cardiovascular Diseases / Q000517:prevention & control*; D002460:Cell Line; D003577:Cytochrome P-450 Enzyme System / Q000378:metabolism*; D011464:Epoprostenol / Q000096:biosynthesis / Q000378:metabolism; D019746:Intramolecular Oxidoreductases; D007535:Isomerases / Q000378:metabolism*; D009131:Muscle, Smooth, Vascular / Q000166:cytology / Q000187:drug effects* / Q000201:enzymology; D013917:Thrombin / Q000494:pharmacology
D016718:Arachidonic Acid; D003577:Cytochrome P-450 Enzyme System; D011464:Epoprostenol; D013917:Thrombin; D007535:Isomerases; D019746:Intramolecular Oxidoreductases; C022461:prostacyclin synthetase; D001241:Aspirin