Atrioventricular node reentry: current concepts and new perspectives.
With the advent of RF catheter modification of AV node conduction for the treatment of AV node reentrant tachycardia, considerable advances have been made with better understanding of the AV junctional anatomy, electrophysiology, and mechanism responsible for AV node reentrant tachycardia. Future studies should be designed to uncover the basic cellular electrophysiological mechanisms responsible for fast and slow AV node conduction, to define the exact tissue components of the reentrant circuit in order to make ablative procedures safer, and to study the long-term effects of RF catheter ablation on AV conduction. Special caution should be directed toward pediatric patients with more stringent indications for catheter ablation of the AV junctional area in these patients.
Clinical Electrophysiology Laboratory, Stanford University Medical Center, California 94305.
R J
RJ
Sung
M R
MR
Lauer
H
H
Chun
D000818:Animals; D001283:Atrioventricular Node / Q000473:pathology / Q000503:physiopathology*; D017115:Catheter Ablation; D004562:Electrocardiography; D004594:Electrophysiology; D006801:Humans; D013611:Tachycardia, Atrioventricular Nodal Reentry / Q000473:pathology / Q000503:physiopathology* / Q000601:surgery