Protein kinase C in signal transduction and carcinogenesis.
Molecular Mechanisms of Tumor Promotion Section, National Cancer Institute, Bethesda, MD.
P M
PM
Blumberg
G
G
Acs
L B
LB
Areces
M G
MG
Kazanietz
N E
NE
Lewin
Z
Z
Szallasi
D016475:3T3 Cells; D000818:Animals; D054713:Bryostatins; D002273:Carcinogens / Q000494:pharmacology; D002471:Cell Transformation, Neoplastic; D004789:Enzyme Activation / Q000187:drug effects; D006801:Humans; D007527:Isoenzymes / Q000502:physiology; D007783:Lactones / Q000494:pharmacology; D018942:Macrolides; D051379:Mice; D008809:Mice, Inbred C3H; D010703:Phorbol Esters / Q000494:pharmacology / Q000633:toxicity; D011485:Protein Binding; D011493:Protein Kinase C / Q000502:physiology*; D011956:Receptors, Cell Surface / Q000502:physiology; D015398:Signal Transduction; D012878:Skin Neoplasms / Q000139:chemically induced; D013329:Structure-Activity Relationship
D054713:Bryostatins; D002273:Carcinogens; D007527:Isoenzymes; D007783:Lactones; D018942:Macrolides; D010703:Phorbol Esters; D011956:Receptors, Cell Surface; C046785:bryostatin 1; C070999:prostratin; D011493:Protein Kinase C