Evidence for the role of the Ah receptor in response to dioxin.
Health Effects Research Laboratory, U.S. Environmental Protection Agency, Research Triangle Park, North Carolina 27711.
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Birnbaum
D000014:Abnormalities, Drug-Induced / Q000235:genetics / Q000378:metabolism; D000483:Alleles; D000818:Animals; D001284:Atrophy; D004147:Dioxins / Q000493:pharmacokinetics / Q000633:toxicity*; D005314:Embryonic and Fetal Development / Q000235:genetics; D007153:Immunologic Deficiency Syndromes / Q000139:chemically induced; D051379:Mice; D008810:Mice, Inbred C57BL; D008811:Mice, Inbred DBA; D018336:Receptors, Aryl Hydrocarbon / Q000187:drug effects / Q000235:genetics / Q000502:physiology*; D013950:Thymus Gland / Q000473:pathology
D004147:Dioxins; D018336:Receptors, Aryl Hydrocarbon