[The acid-forming function of the stomach and the signs of chronic "restructuring" gastritis in patients with pulmonary tuberculosis combined with clonorchiasis or metagonimiasis].
The study has shown that 100% of patients with pulmonary tuberculosis concurrent with clonorchiasis and metagonimiasis develop a symptomatology of type A chronic gastritis with sharply inhibited gastric acid-producing function. The morphological features of this chronic atrophic gastritis are formation of surface microerosions and abrupt mucoidization of tegmental, foveolate, and cervical epitheliocytes.
Korablev
V N
VN
Koval'skiĭ
G B
GB
D000818:Animals; D001706:Biopsy; D002908:Chronic Disease; D003003:Clonorchiasis / Q000150:complications / Q000503:physiopathology*; D005744:Gastric Acid / Q000378:metabolism*; D005745:Gastric Acidity Determination; D005753:Gastric Mucosa / Q000378:metabolism / Q000473:pathology; D005756:Gastritis / Q000209:etiology / Q000503:physiopathology*; D006578:Heterophyidae; D006632:Histamine; D006801:Humans; D014201:Trematode Infections / Q000150:complications / Q000503:physiopathology*; D014397:Tuberculosis, Pulmonary / Q000150:complications / Q000503:physiopathology*
D006632:Histamine
Kislotoobrazuiushchaia funktsiia zheludka i priznaki khronicheskogo gastrita "perestroĭki" u bol'nykh tuberkulezom legkikh v sochetanii s klonorkhozom ili metagonimozom.