[Current findings on certain physiopathological aspects of the inappropriate secretion of antidiuretic hormone syndrome].
Hyponatraemia secondary to inappropriate secretion of antidiuretic hormone (SIADH) induces some biological modifications which are relatively characteristic of this condition. The disturbed acid-base equilibrium and the mechanism involved in the high urea and uric acid clearances observed in SIADH are discussed.
Service de Médecine interne, Hôpital Erasme, Bruxelles.
G
G
Decaux
B
B
Namias
A
A
Soupart
D000137:Acid-Base Imbalance / Q000378:metabolism; D005919:Glomerular Filtration Rate; D006801:Humans; D007010:Hyponatremia / Q000503:physiopathology; D007177:Inappropriate ADH Syndrome / Q000503:physiopathology*; D011086:Polycythemia / Q000503:physiopathology; D014508:Urea / Q000097:blood; D014527:Uric Acid / Q000097:blood
D014527:Uric Acid; D014508:Urea
Données récentes sur certains aspects de la physiopathologie du syndrome de sécrétion inappropriée d'hormone anti-diurétique.