Mutations in the p53 tumor suppressor gene: clues to cancer etiology and molecular pathogenesis.
Greenblatt
M S
MS
Laboratory of Human Carcinogenesis, National Cancer Institute, Bethesda, Maryland 20892.
Bennett
W P
WP
Hollstein
M
M
Harris
C C
CC
D000348:Aflatoxins / Q000633:toxicity; D000818:Animals; D002471:Cell Transformation, Neoplastic / Q000235:genetics; D002872:Chromosome Deletion; D017124:Conserved Sequence; D004249:DNA Damage; D004252:DNA Mutational Analysis; D004260:DNA Repair; D004261:DNA Replication; D004195:Disease Models, Animal; D016158:Genes, p53 / Q000235:genetics* / Q000502:physiology; D006509:Hepatitis B / Q000150:complications; D006801:Humans; D009154:Mutation / Q000235:genetics*; D009369:Neoplasms / Q000209:etiology / Q000235:genetics*; D009381:Neoplasms, Radiation-Induced / Q000235:genetics; D010641:Phenotype; D017422:Sequence Analysis, DNA; D012907:Smoking / Q000009:adverse effects; D013329:Structure-Activity Relationship; D016159:Tumor Suppressor Protein p53 / Q000737:chemistry / Q000502:physiology
D000348:Aflatoxins; D016159:Tumor Suppressor Protein p53