Induction of a neuroprotective state in cerebellar granule cells following activation of N-methyl-D-aspartate receptors.
Marini
A M
AM
Section on Molecular Pharmacology, National Institutes of Health, National Institute of Mental Health, Bethesda, Maryland 20892.
Paul
S M
SM
D000818:Animals; D002470:Cell Survival / Q000187:drug effects; D002478:Cells, Cultured; D002531:Cerebellum / Q000187:drug effects* / Q000378:metabolism / Q000473:pathology; D003513:Cycloheximide / Q000494:pharmacology; D003609:Dactinomycin / Q000494:pharmacology; D016291:Dizocilpine Maleate / Q000494:pharmacology; D005971:Glutamates / Q000494:pharmacology* / Q000633:toxicity; D018698:Glutamic Acid; D016202:N-Methylaspartate / Q000494:pharmacology* / Q000633:toxicity; D009474:Neurons / Q000187:drug effects* / Q000378:metabolism / Q000473:pathology; D009498:Neurotoxins / Q000633:toxicity*; D011726:Pyridinium Compounds / Q000037:antagonists & inhibitors / Q000633:toxicity*; D016194:Receptors, N-Methyl-D-Aspartate / Q000187:drug effects / Q000378:metabolism*
D005971:Glutamates; D009498:Neurotoxins; D011726:Pyridinium Compounds; D016194:Receptors, N-Methyl-D-Aspartate; D003609:Dactinomycin; D018698:Glutamic Acid; C026560:1-(4-methoxyphenyl)pyridinium; D016202:N-Methylaspartate; D016291:Dizocilpine Maleate; D003513:Cycloheximide