How cells respond to stress.
During emergencies, cells produce stress proteins that repair damage. Inquiry into how they work offers promise for coping with infection, autoimmune disease and even cancer.
Welch
W J
WJ
Lung Biology Center, University of California, San Francisco.
D000255:Adenosine Triphosphate / Q000378:metabolism; D000818:Animals; D001327:Autoimmune Diseases; D001419:Bacteria / Q000235:genetics; D018407:Bacterial Physiological Phenomena; D001426:Bacterial Proteins / Q000235:genetics / Q000378:metabolism; D002468:Cell Physiological Phenomena; D018835:Chaperonin 10; D018834:Chaperonin 60; D004331:Drosophila melanogaster / Q000235:genetics / Q000254:growth & development; D015870:Gene Expression; D006360:Heat-Shock Proteins / Q000235:genetics / Q000378:metabolism / Q000502:physiology*; D006358:Hot Temperature; D006801:Humans; D007239:Infections; D009369:Neoplasms; D011956:Receptors, Cell Surface / Q000502:physiology; D013312:Stress, Physiological
D001426:Bacterial Proteins; D018835:Chaperonin 10; D018834:Chaperonin 60; D006360:Heat-Shock Proteins; D011956:Receptors, Cell Surface; D000255:Adenosine Triphosphate