[Pathology of muscle contracture caused by injection and activity of phosphochesterase and insulin receptor].
The animal model of muscle contracture caused by injection was set up in the experiment. We used the model for excessive healing studies and researched the changes in its pathology, and activity of phosphochesterase and insulin receptor in this model. The experimental results revealed that the pathologic changes belonged to excessive proliferation of connective tissue (excessive healing). When occurred muscle degeneration, infiltration of inflammatory cells, proliferation of fibroblasts and descending of collagen fibers, the activity level phosphochesterase and insulin receptor became higher. The excessive healing occurrence may have something to do with the changes of phosphochesterase and insulin receptor.
Liu
M
M
Peng
M
M
Hu
T
T
Chen
M
M
Lang
S
S
D015720:5'-Nucleotidase / Q000378:metabolism*; D000818:Animals; D002462:Cell Membrane / Q000378:metabolism; D003286:Contracture / Q000209:etiology / Q000378:metabolism / Q000473:pathology*; D007273:Injections, Intramuscular / Q000009:adverse effects*; D011817:Rabbits; D011972:Receptor, Insulin / Q000378:metabolism*
D011972:Receptor, Insulin; D015720:5'-Nucleotidase