title: Respiratory function in workers employed in the glassblowing industry.
abstract: A group of 80 men employed in the glassblowing industry was studied in order to investigate the effect of this occupational exposure on respiratory function. Eighty nonexposed workers were included in the study as a control group. Glassblowers had a significantly higher prevalence of chronic bronchitis, nasal catarrh, chronic sinusitis, and nasal bleeding than control workers; length of employment in the industry did not affect the prevalence of symptoms. Many of the glassblowers complained of work shift related symptoms. Measurement of lung function among glassblowers showed there were significant increases in the forced vital capacity (FVC) and the maximum flow rates at 50% and 25% of FVC on maximum expiratory flow volume (MEFV) curves (FEF50, FEF25) across the work shift. Glassblowers had significantly larger preshift FVC and forced expiratory volume in 1-second (FEV1) measurements when compared to controls. Additionally, residual volume (RV) and RV/TLC% for the glassblowers were significantly increased while the diffusing capacity (DLCO) was normal (when compared to predicted values). Our data indicate that employment in the glassblowing industry contributes to the development of chronic respiratory findings.
authors.affiliation: Andrija Stampar School of Public Health, Zagreb. Croatia.
authors.forename: E
authors.initials: E
authors.lastname: Zuskin
authors.forename: D
authors.initials: D
authors.lastname: Butkovic
authors.forename: E N
authors.initials: EN
authors.lastname: Schachter
authors.forename: J
authors.initials: J
authors.lastname: Mustajbegovic
mesh_terms: D000328:Adult; D002908:Chronic Disease; D005898:Glass; D006801:Humans; D007221:Industry; D008168:Lung / Q000502:physiology*; D008297:Male; D008875:Middle Aged; D009784:Occupational Diseases / Q000209:etiology* / Q000503:physiopathology; D016273:Occupational Exposure / Q000009:adverse effects*; D012129:Respiratory Function Tests; D012140:Respiratory Tract Diseases / Q000209:etiology* / Q000503:physiopathology