[The behavioral consequences of prenatal hemic hypoxia in rat progeny].
The prenatal hemic hypoxia was caused by methemoglobin inducing agent, sodium nitrite, injected intraperitoneally in different doses from 10th to 19th day of pregnancy. It was shown that perinatal hemic hypoxia evoked delayed neurological deficit (disorders of motor coordination, hypodynamia) and disturbances of mnestic functions (impairment of learning and memory in passive and active avoidance paradigms and in the test of extrapolative water escape). The most pronounced disorders of CNS functions were shown in offsprings of male rats injected with sodium nitrite in a dose of 40 mg/kg.
Koziar
V S
VS
Trofimov
S S
SS
Ostrovskaia
R U
RU
Sariev
A K
AK
Zherdev
V P
VP
D000818:Animals; D001522:Behavior, Animal / Q000502:physiology*; D002490:Central Nervous System / Q000503:physiopathology; D002908:Chronic Disease; D003214:Conditioning, Classical / Q000502:physiology; D004195:Disease Models, Animal; D005260:Female; D000860:Hypoxia / Q000139:chemically induced / Q000503:physiopathology*; D008297:Male; D011247:Pregnancy; D011297:Prenatal Exposure Delayed Effects; D051381:Rats; D017208:Rats, Wistar; D012727:Sex Characteristics; D012977:Sodium Nitrite
D012977:Sodium Nitrite
Povedencheskie posledstviia prenatal'noĭ gemicheskoĭ gipoksii u potomstva krys.