Absence of enhancement of spontaneous echocardiographic contrast by thrombocytosis in a patient with left ventricular aneurysm, primary thrombocythemia, and von Recklinghausen neurofibromatosis: a case report.
A thirty-eight-year-old man with primary thrombocythemia, von Recklinghausen neurofibromatosis, and myocardial-infarction-related left ventricular aneurysm with spontaneous echocardiographic contrast was followed up, suggesting that: 1. Neurofibromatosis may promote silent myocardial infarction or ischemia. Whether involvement of cardiac sensory nerves is a possible underlying mechanism remains nevertheless uncertain. 2. Platelets, whose role in the genesis of spontaneous echocardiographic contrast has been advocated, are probably not involved in this phenomenon, even in large numbers.
Monsuez
J J
JJ
Intensive Care Unit, Hôpital Saint-Louis, Paris, France.
Janier
M
M
Van-Cao
A N
AN
Le Gall
J R
JR
D000328:Adult; D004452:Echocardiography; D004562:Electrocardiography; D006322:Heart Aneurysm / Q000097:blood / Q000000981:diagnostic imaging* / Q000209:etiology; D006352:Heart Ventricles / Q000000981:diagnostic imaging; D006801:Humans; D008297:Male; D009203:Myocardial Infarction / Q000097:blood / Q000150:complications / Q000000981:diagnostic imaging; D009456:Neurofibromatosis 1 / Q000097:blood / Q000000981:diagnostic imaging*; D010974:Platelet Aggregation; D010976:Platelet Count; D013920:Thrombocythemia, Essential / Q000097:blood / Q000000981:diagnostic imaging*; D013922:Thrombocytosis / Q000097:blood / Q000000981:diagnostic imaging*