title: [Reduced "oxidative burst" in a granulocyte subpopulation in a case of Sweet syndrome].
abstract: Oxidative burst was defective in 50% of peripheral blood neutrophils in a case of Sweet syndrome with leukemoid granulocytosis. Phagocytosis was normal. We suggest that the decreased ability to produce oxygen radicals observed in this patient might lead to a compensatory recruitment of hematopoietic growth factors. Consecutive activation, increased chemotaxis and adhesion of polymorphonuclear granulocytes might be the cause of the neutrophilic dermal infiltrate of Sweet syndrome.
authors.affiliation: Abteilung Rhematologie, Universität Freiburg.
authors.forename: U A
authors.initials: UA
authors.lastname: Walker
authors.forename: B
authors.initials: B
authors.lastname: Riecken
authors.forename: E W
authors.initials: EW
authors.lastname: Herbst
authors.forename: G
authors.initials: G
authors.lastname: Wolff-Vorbeck
authors.forename: M
authors.initials: M
authors.lastname: Schlesier
authors.forename: H H
authors.initials: HH
authors.lastname: Peter
mesh_terms: D000328:Adult; D005260:Female; D006098:Granulocytes / Q000502:physiology*; D006801:Humans; D009504:Neutrophils / Q000502:physiology; D016897:Respiratory Burst; D016463:Sweet Syndrome / Q000503:physiopathology*
vernacular_title: Verminderter "oxidative burst" in einer Granulozytensubpopulation bei einem Fall von Sweet-Syndrom.