The hypercoagulable states.
In most cases, acute venous thrombosis has a straightforward etiology--for example, the patient who has had prolonged bed rest with a broken leg and received no heparin. A significant minority of patients, however, have an inherited biochemical deficiency or an acquired disorder that increases the risk of thrombosis. Some general guidelines suggest when to suspect such defects and what tests to apply.
Alving
B M
BM
Department of Hematology, Walter Reed Army Institute of Research, Washington, D.C.
D000293:Adolescent; D000328:Adult; D000367:Age Factors; D000368:Aged; D016736:Antiphospholipid Syndrome / Q000150:complications; D000990:Antithrombin III / Q000032:analysis; D020152:Antithrombin III Deficiency; D001777:Blood Coagulation / Q000502:physiology; D001778:Blood Coagulation Disorders / Q000097:blood / Q000175:diagnosis / Q000209:etiology; D002985:Clinical Protocols / Q000592:standards; D003937:Diagnosis, Differential; D030342:Genetic Diseases, Inborn / Q000097:blood / Q000175:diagnosis; D006801:Humans; D008875:Middle Aged; D009369:Neoplasms / Q000150:complications; D011486:Protein C / Q000032:analysis; D020151:Protein C Deficiency; D017293:Protein S / Q000097:blood; D018455:Protein S Deficiency; D013927:Thrombosis / Q000097:blood / Q000175:diagnosis / Q000209:etiology
D011486:Protein C; D017293:Protein S; D000990:Antithrombin III