Sodium fluoride induced activation of phospholipase C in intact human platelets does not depend on ADP, PAF or arachidonate products.
Pfliegler
G
G
Center for Thrombosis and Vascular Research, University of Leuven, Belgium.
Arnout
J
J
Kienast
J
J
Wittevrongel
C
C
Hoet
B
B
Deckmyn
H
H
Vermylen
J
J
D000244:Adenosine Diphosphate / Q000378:metabolism; D001241:Aspirin / Q000031:analogs & derivatives / Q000378:metabolism; D001792:Blood Platelets / Q000201:enzymology*; D004789:Enzyme Activation / Q000187:drug effects; D019204:GTP-Binding Proteins / Q000187:drug effects*; D006801:Humans; D008239:Lysine / Q000031:analogs & derivatives / Q000378:metabolism; D010742:Phospholipid Ethers / Q000494:pharmacology; D010972:Platelet Activating Factor / Q000037:antagonists & inhibitors*; D012969:Sodium Fluoride / Q000494:pharmacology*; D010738:Type C Phospholipases / Q000096:biosynthesis* / Q000187:drug effects
D010742:Phospholipid Ethers; D010972:Platelet Activating Factor; D000244:Adenosine Diphosphate; C037913:CV 3988; D012969:Sodium Fluoride; D010738:Type C Phospholipases; D019204:GTP-Binding Proteins; D008239:Lysine; D001241:Aspirin; C010395:acetylsalicylic acid lysinate