Heat shock proteins used to show that haloperidol prevents neuronal injury produced by ketamine, MK801, and phencyclidine.
Department of Neurology, University of California, San Francisco 94117.
F R
FR
Sharp
M
M
Butman
S
S
Wang
J
J
Koistinaho
S H
SH
Graham
S M
SM
Sagar
P
P
Berger
F M
FM
Longo
D000818:Animals; D015415:Biomarkers; D016923:Cell Death / Q000187:drug effects; D016291:Dizocilpine Maleate / Q000633:toxicity*; D004305:Dose-Response Relationship, Drug; D006179:Gyrus Cinguli / Q000187:drug effects* / Q000473:pathology; D006220:Haloperidol / Q000494:pharmacology*; D006360:Heat-Shock Proteins / Q000032:analysis / Q000096:biosynthesis*; D007649:Ketamine / Q000633:toxicity*; D009474:Neurons / Q000166:cytology / Q000187:drug effects* / Q000473:pathology; D009498:Neurotoxins / Q000633:toxicity*; D010622:Phencyclidine / Q000633:toxicity*; D051381:Rats
D015415:Biomarkers; D006360:Heat-Shock Proteins; D009498:Neurotoxins; D007649:Ketamine; D016291:Dizocilpine Maleate; D010622:Phencyclidine; D006220:Haloperidol