B-50/GAP-43 expression via a defective herpes simplex virus vector results in persistent morphological changes in non-neuronal cells.
Rudolf Magnus Institute, Department of Pharmacology, Utrecht, The Netherlands.
J
J
Verhaagen
W T
WT
Hermens
M G
MG
Kaplitt
D W
DW
Pfaff
W H
WH
Gispen
D000818:Animals; D002478:Cells, Cultured; D002522:Chlorocebus aethiops; D003587:Cytomegalovirus / Q000235:genetics; D003673:Defective Viruses / Q000235:genetics*; D019922:GAP-43 Protein; D018014:Gene Transfer Techniques; D005822:Genetic Vectors; D006133:Growth Substances / Q000235:genetics*; D018259:Herpesvirus 1, Human / Q000235:genetics*; D007150:Immunohistochemistry; D008562:Membrane Glycoproteins / Q000235:genetics*; D009419:Nerve Tissue Proteins / Q000235:genetics*; D011401:Promoter Regions, Genetic; D011817:Rabbits; D014709:Vero Cells; D001616:beta-Galactosidase / Q000235:genetics
D019922:GAP-43 Protein; D006133:Growth Substances; D008562:Membrane Glycoproteins; D009419:Nerve Tissue Proteins; D001616:beta-Galactosidase