The three processes leading to post PTCA restenosis: dependence on the lesion substrate.
Fuster
V
V
Cardiovascular Biology Research Laboratory, Cardiovascular Institute Mount Sinai School of Medicine, New York, NY 10029, USA.
Falk
E
E
Fallon
J T
JT
Badimon
L
L
Chesebro
J H
JH
Badimon
J J
JJ
D015906:Angioplasty, Balloon, Coronary / Q000009:adverse effects*; D001161:Arteriosclerosis; D003324:Coronary Artery Disease / Q000473:pathology / Q000517:prevention & control / Q000628:therapy*; D016503:Drug Delivery Systems; D004730:Endothelium, Vascular / Q000473:pathology; D005109:Extracellular Matrix / Q000378:metabolism; D005343:Fibrinolytic Agents / Q000627:therapeutic use; D005786:Gene Expression Regulation; D006133:Growth Substances / Q000502:physiology; D006331:Heart Diseases / Q000209:etiology / Q000628:therapy; D006801:Humans; D008264:Macrophages / Q000473:pathology; D008954:Models, Biological; D009131:Muscle, Smooth, Vascular / Q000473:pathology; D012008:Recurrence; D015607:Stents; D013917:Thrombin / Q000378:metabolism; D013927:Thrombosis / Q000209:etiology / Q000628:therapy
D005343:Fibrinolytic Agents; D006133:Growth Substances; D013917:Thrombin