Promoting mechanisms of CO2 on neuroendocrine cell proliferation mediated by nicotinic receptor stimulation. Significance for lung cancer risk in individuals with chronic lung disease.
Schuller
H M
HM
The Carcinogenesis and Developmental Therapeutics Program, College of Veterinary Medicine, University of Tennessee, Knoxville 37996-4500, USA.
Miller
M S
MS
Park
P G
PG
Orloff
M S
MS
D000818:Animals; D002245:Carbon Dioxide / Q000502:physiology*; D018278:Carcinoma, Neuroendocrine / Q000378:metabolism* / Q000473:pathology; D002455:Cell Division; D002908:Chronic Disease; D006224:Cricetinae; D006801:Humans; D008171:Lung Diseases / Q000378:metabolism; D008175:Lung Neoplasms / Q000378:metabolism* / Q000473:pathology; D011978:Receptors, Nicotinic / Q000502:physiology*; D014407:Tumor Cells, Cultured
D011978:Receptors, Nicotinic; D002245:Carbon Dioxide