Potential mechanism of abnormal thromboregulation in xenograft rejection: loss of ecto-ATPases upon endothelial cell activation.
Robson
S C
SC
Sandoz Center for Immunobiology, New England Deaconess Hospital, Harvard University, Boston, Massachusetts 02215, USA.
Candinas
D
D
Siegel
J B
JB
Kopp
C
C
Millan
M
M
Hancock
W W
WW
Bach
F H
FH
D000244:Adenosine Diphosphate / Q000494:pharmacology; D000251:Adenosine Triphosphatases / Q000037:antagonists & inhibitors / Q000378:metabolism*; D000818:Animals; D002478:Cells, Cultured; D004730:Endothelium, Vascular / Q000502:physiology* / Q000503:physiopathology; D006084:Graft Rejection / Q000276:immunology / Q000503:physiopathology*; D006801:Humans; D015539:Platelet Activation / Q000187:drug effects; D010974:Platelet Aggregation / Q000187:drug effects; D011994:Recombinant Proteins / Q000494:pharmacology; D013552:Swine; D014183:Transplantation, Heterologous / Q000276:immunology / Q000502:physiology*
D011994:Recombinant Proteins; D000244:Adenosine Diphosphate; D000251:Adenosine Triphosphatases; C027201:ectoATPase