How beneficial is the reduction of edema formation by polyethylene glycol during cardioplegic arrest?
Kober
I M
IM
Institute of Medical Physiology, University of Vienna, Austria.
Obermayr
R P
RP
Spieckermann
P G
PG
D000818:Animals; D001794:Blood Pressure / Q000187:drug effects; D002314:Cardioplegic Solutions; D003326:Coronary Circulation / Q000187:drug effects; D004487:Edema / Q000517:prevention & control*; D005947:Glucose; D006321:Heart / Q000187:drug effects / Q000502:physiology* / Q000503:physiopathology; D006323:Heart Arrest; D006339:Heart Rate / Q000187:drug effects; D006439:Hemodynamics / Q000187:drug effects*; D066298:In Vitro Techniques; D008297:Male; D008353:Mannitol; D017202:Myocardial Ischemia / Q000503:physiopathology; D015425:Myocardial Reperfusion / Q000379:methods; D011092:Polyethylene Glycols / Q000494:pharmacology*; D011189:Potassium Chloride; D011343:Procaine; D051381:Rats; D017207:Rats, Sprague-Dawley; D014325:Tromethamine
C039984:Bretschneider cardioplegic solution; D002314:Cardioplegic Solutions; C074097:Krebs-Henseleit solution; D014325:Tromethamine; D008353:Mannitol; D011092:Polyethylene Glycols; D011343:Procaine; D011189:Potassium Chloride; D005947:Glucose