DT-diaphorase induction by lead acetate in the liver of rats.
Arizono
K
K
Department of Hygienic Chemistry and Toxicology, Faculty of Pharmaceutical Sciences, Nagasaki University, 1-14 Bunkyo-cho, Nagasaki 852, Japan.
Sugiura
S
S
Miyazato
S
S
Takiguchi
M
M
Ariyoshi
T
T
D015086:2,6-Dichloroindophenol / Q000378:metabolism; D000818:Animals; D002121:Calcium Channel Blockers / Q000494:pharmacology; D003609:Dactinomycin / Q000494:pharmacology; D004110:Diltiazem / Q000494:pharmacology; D004305:Dose-Response Relationship, Drug; D004790:Enzyme Induction / Q000187:drug effects; D007274:Injections, Intraperitoneal; D007668:Kidney / Q000187:drug effects / Q000201:enzymology; D007854:Lead / Q000008:administration & dosage / Q000633:toxicity*; D008099:Liver / Q000187:drug effects* / Q000201:enzymology; D008297:Male; D009252:NADPH Dehydrogenase / Q000096:biosynthesis / Q000378:metabolism*; D009942:Organometallic Compounds / Q000008:administration & dosage / Q000633:toxicity*; D014176:Protein Biosynthesis; D011500:Protein Synthesis Inhibitors / Q000494:pharmacology; D051381:Rats; D017208:Rats, Wistar; D013056:Spectrophotometry, Ultraviolet
D002121:Calcium Channel Blockers; D009942:Organometallic Compounds; D011500:Protein Synthesis Inhibitors; D003609:Dactinomycin; D007854:Lead; D015086:2,6-Dichloroindophenol; D009252:NADPH Dehydrogenase; D004110:Diltiazem; C008261:lead acetate