[The interrelationships of the hypothalamo-neurohypophyseal, renin-angiotensin-aldosterone and prostanoid systems i regulating central hemodynamics in the acute period of traumatic disease].
Experiments on white male rats with a traumatic shock by the Cannon method have permitted establishing that expressed activation of hypothalamo-neurohypophysial, renin-angiotensin-aldosterone and prostanoid systems induced disorders in the cardiovascular system, namely, a decrease in the heart stroke, growth of the vascular tension in the first hours of the acute period of the disease and an increase in the peripheral vascular resistance. Besides, the renin plasma activity falls, which may be a sanogenesis mechanism preventing redundant stimulation of the vessels and myocardium.
Barinov
E F
EF
Ziablitsev
S V
SV
D000208:Acute Disease; D000704:Analysis of Variance; D000818:Animals; D006439:Hemodynamics / Q000502:physiology*; D007030:Hypothalamo-Hypophyseal System / Q000503:physiopathology*; D008297:Male; D010904:Pituitary Gland, Posterior / Q000503:physiopathology; D011453:Prostaglandins / Q000502:physiology*; D051381:Rats; D012044:Regression Analysis; D012084:Renin-Angiotensin System / Q000502:physiology*; D012774:Shock, Traumatic / Q000097:blood / Q000503:physiopathology; D013997:Time Factors; D014947:Wounds and Injuries / Q000097:blood / Q000503:physiopathology*
D011453:Prostaglandins