Ribonuclease H-mediated antisense effects in intact human leukaemia cells.
Tidd
D M
DM
Department of Biochemistry, University of Liverpool, U.K.
D001693:Biological Transport, Active; D016044:Fusion Proteins, bcr-abl / Q000235:genetics; D015972:Gene Expression Regulation, Neoplastic; D018390:Gene Targeting; D006801:Humans; D007938:Leukemia / Q000235:genetics / Q000378:metabolism / Q000628:therapy*; D015464:Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Q000235:genetics / Q000378:metabolism / Q000628:therapy; D015394:Molecular Structure; D016376:Oligonucleotides, Antisense / Q000737:chemistry / Q000378:metabolism* / Q000494:pharmacology*; D009857:Oncogenes; D017354:Point Mutation; D016914:Ribonuclease H / Q000378:metabolism*; D013489:Suppression, Genetic; D014407:Tumor Cells, Cultured
D016376:Oligonucleotides, Antisense; D016044:Fusion Proteins, bcr-abl; D016914:Ribonuclease H