Trans-activation of cellular genes by hepatitis B virus proteins: a possible mechanism of hepatocarcinogenesis.
Department of Medicine II, Klinikum Grosshadern, Ludwig-Maximilians-Universität, Munich, Germany.
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Caselmann
D000818:Animals; D006528:Carcinoma, Hepatocellular / Q000821:virology; D002477:Cells; D005544:Forecasting; D005796:Genes; D006509:Hepatitis B / Q000821:virology*; D006514:Hepatitis B Surface Antigens / Q000378:metabolism*; D006515:Hepatitis B virus / Q000235:genetics / Q000378:metabolism* / Q000648:ultrastructure; D006801:Humans; D015534:Trans-Activators / Q000378:metabolism*; D015533:Transcriptional Activation; D054334:Viral Regulatory and Accessory Proteins
D006514:Hepatitis B Surface Antigens; D015534:Trans-Activators; D054334:Viral Regulatory and Accessory Proteins; C057628:hepatitis B virus X protein