title: [Mechanism of action of electromagnetic fields biologically adequate to man].
abstract: The paper analyzes 10-year experience in making experimental observations and clinically using electromagnetic fields (EMF) biologically adequate to man. Basic investigations of the electromechanical process that lead to the occurrence of currents in the bone tissue and blood vascular walls in their functional deformation were conducted. They showed that the piezoelectric effect is the most marked in the intact bone tissue. Experimental studies determined the amplitude and shape of impulses of currents emerging in the intact bone tissue of the extremities and in the walls of blood vessels during pulse blood filling. The bioelectric tissue metabolism changes in various human locomotor abnormalities. The lost part in the diseased area can be restored by means of external EMF. On this basis, devices for human biologically adequate electromagnetic therapy were designed. The efficiency of each session of electromagnetic therapy was evaluated from the normalization of blood circulation in the diseased area. The efficiency of this therapy in vascular disease was noted to depend on the synchronization of the generated electromagnetic pulse with the pulse wave of a patient and its phases. An experiment on 13 intact mongrel dogs was made to reveal the impact of EMF biologically adequate to man on bone marrow hemopoiesis. EMF 2 was found to have a stimulating effect on granulocytopoiesis. Platelet counts were elevated in the peripheral blood of animals. Analysing 2000 clinical cases and canine experimental findings provided evidence for the following mechanism of action of the electromagnetic fields biologically adequate to man. The fields exert a primary initial and virtually rapid action on the active areas of nerve structures and smooth muscle of blood vascular walls. They show a primary, but slower action on blood cells and on the vascular endothelial structure. There may be a secondary impact of EMF on some endocrine glands, which causes changes in the protein metabolism of a biological object and appears as stimulation of granulocytopoiesis and depot platelet release into the peripheral blood stream.
authors.forename: S A
authors.initials: SA
authors.lastname: Schastnyĭ
authors.forename: S I
authors.initials: SI
authors.lastname: Shchukin
authors.forename: I M
authors.initials: IM
authors.lastname: Roslyĭ
authors.forename: V G
authors.initials: VG
authors.lastname: Zubenko
authors.forename: K R
authors.initials: KR
authors.lastname: Beliaev
authors.forename: G I
authors.initials: GI
authors.lastname: Semikin
authors.forename: A A
authors.initials: AA
authors.lastname: Morozov
mesh_terms: D000328:Adult; D000818:Animals; D002648:Child; D004285:Dogs; D004574:Electromagnetic Fields; D006801:Humans; D008280:Magnetics / Q000627:therapeutic use*; D008954:Models, Biological; D013997:Time Factors