Interleukin-12 gene expression mediates the accelerating effect of cyclophosphamide in autoimmune disease.
Rothe
H
H
Diabetes Research Institute, Heinrich-Heine University of Düsseldorf, Germany.
Burkart
V
V
Faust
A
A
Kolb
H
H
D000818:Animals; D001327:Autoimmune Diseases / Q000503:physiopathology*; D003520:Cyclophosphamide / Q000494:pharmacology*; D003922:Diabetes Mellitus, Type 1 / Q000503:physiopathology; D005260:Female; D015870:Gene Expression / Q000187:drug effects; D007371:Interferon-gamma / Q000235:genetics; D018664:Interleukin-12 / Q000235:genetics*; D007515:Islets of Langerhans / Q000503:physiopathology; D051379:Mice; D008807:Mice, Inbred BALB C; D016688:Mice, Inbred NOD; D013154:Spleen / Q000166:cytology / Q000503:physiopathology; D018417:Th1 Cells / Q000276:immunology; D018418:Th2 Cells / Q000276:immunology; D014409:Tumor Necrosis Factor-alpha / Q000235:genetics
D014409:Tumor Necrosis Factor-alpha; D018664:Interleukin-12; D007371:Interferon-gamma; D003520:Cyclophosphamide